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Heart-resident CCR2.

JCI insight

Li W, Hsiao HM, Higashikubo R, Saunders BT, Bharat A, Goldstein DR, Krupnick AS, Gelman AE, Lavine KJ, Kreisel D.
PMID: 27536731
JCI Insight. 2016 Aug 04;1(12). doi: 10.1172/jci.insight.87315.

It is well established that maladaptive innate immune responses to sterile tissue injury represent a fundamental mechanism of disease pathogenesis. In the context of cardiac ischemia reperfusion injury, neutrophils enter inflamed heart tissue, where they play an important role...

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Li W, Hsiao HM, Higashikubo R, et al. Heart-resident CCR2. JCI Insight. 2016;1(12)doi: 10.1172/jci.insight.87315.
Li, W., Hsiao, H. M., Higashikubo, R., Saunders, B. T., Bharat, A., Goldstein, D. R., Krupnick, A. S., Gelman, A. E., Lavine, K. J., & Kreisel, D. (2016). Heart-resident CCR2. JCI insight, 1(12), . https://doi.org/10.1172/jci.insight.87315
Li, Wenjun, et al. "Heart-resident CCR2." JCI insight vol. 1,12 (2016). doi: https://doi.org/10.1172/jci.insight.87315
Li W, Hsiao HM, Higashikubo R, Saunders BT, Bharat A, Goldstein DR, Krupnick AS, Gelman AE, Lavine KJ, Kreisel D. Heart-resident CCR2. JCI Insight. 2016 Aug 04;1(12). doi: 10.1172/jci.insight.87315. PMID: 27536731; PMCID: PMC4985028.
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IRF5 distinguishes severe asthma in humans and drives Th1 phenotype and airway hyperreactivity in mice.

JCI insight

Oriss TB, Raundhal M, Morse C, Huff RE, Das S, Hannum R, Gauthier MC, Scholl KL, Chakraborty K, Nouraie SM, Wenzel SE, Ray P, Ray A.
PMID: 28515358
JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.91019. eCollection 2017 May 18.

Severe asthma (SA) is a significant problem both clinically and economically, given its poor response to corticosteroids (CS). We recently reported a complex type 1-dominated (IFN-γ-dominated) immune response in more than 50% of severe asthmatics despite high-dose CS treatment....

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Oriss TB, Raundhal M, Morse C, et al. IRF5 distinguishes severe asthma in humans and drives Th1 phenotype and airway hyperreactivity in mice. JCI Insight. 2017;2(10)doi: 10.1172/jci.insight.91019.
Oriss, T. B., Raundhal, M., Morse, C., Huff, R. E., Das, S., Hannum, R., Gauthier, M. C., Scholl, K. L., Chakraborty, K., Nouraie, S. M., Wenzel, S. E., Ray, P., & Ray, A. (2017). IRF5 distinguishes severe asthma in humans and drives Th1 phenotype and airway hyperreactivity in mice. JCI insight, 2(10), . https://doi.org/10.1172/jci.insight.91019
Oriss, Timothy B, et al. "IRF5 distinguishes severe asthma in humans and drives Th1 phenotype and airway hyperreactivity in mice." JCI insight vol. 2,10 (2017). doi: https://doi.org/10.1172/jci.insight.91019
Oriss TB, Raundhal M, Morse C, Huff RE, Das S, Hannum R, Gauthier MC, Scholl KL, Chakraborty K, Nouraie SM, Wenzel SE, Ray P, Ray A. IRF5 distinguishes severe asthma in humans and drives Th1 phenotype and airway hyperreactivity in mice. JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.91019. eCollection 2017 May 18. PMID: 28515358; PMCID: PMC5436536.
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T cell progenitor therapy-facilitated thymopoiesis depends upon thymic input and continued thymic microenvironment interaction.

JCI insight

Smith MJ, Reichenbach DK, Parker SL, Riddle MJ, Mitchell J, Osum KC, Mohtashami M, Stefanski HE, Fife BT, Bhandoola A, Hogquist KA, Holländer GA, Zúñiga-Pflücker JC, Tolar J, Blazar BR.
PMID: 28515359
JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.92056. eCollection 2017 May 18.

Infusion of in vitro-derived T cell progenitor (proT) therapy with hematopoietic stem cell transplant aids the recovery of the thymus damaged by total body irradiation. To understand the interaction between proTs and the thymic microenvironment, WT mice were lethally...

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Smith MJ, Reichenbach DK, Parker SL, et al. T cell progenitor therapy-facilitated thymopoiesis depends upon thymic input and continued thymic microenvironment interaction. JCI Insight. 2017;2(10)doi: 10.1172/jci.insight.92056.
Smith, M. J., Reichenbach, D. K., Parker, S. L., Riddle, M. J., Mitchell, J., Osum, K. C., Mohtashami, M., Stefanski, H. E., Fife, B. T., Bhandoola, A., Hogquist, K. A., Holländer, G. A., Zúñiga-Pflücker, J. C., Tolar, J., & Blazar, B. R. (2017). T cell progenitor therapy-facilitated thymopoiesis depends upon thymic input and continued thymic microenvironment interaction. JCI insight, 2(10), . https://doi.org/10.1172/jci.insight.92056
Smith, Michelle J, et al. "T cell progenitor therapy-facilitated thymopoiesis depends upon thymic input and continued thymic microenvironment interaction." JCI insight vol. 2,10 (2017). doi: https://doi.org/10.1172/jci.insight.92056
Smith MJ, Reichenbach DK, Parker SL, Riddle MJ, Mitchell J, Osum KC, Mohtashami M, Stefanski HE, Fife BT, Bhandoola A, Hogquist KA, Holländer GA, Zúñiga-Pflücker JC, Tolar J, Blazar BR. T cell progenitor therapy-facilitated thymopoiesis depends upon thymic input and continued thymic microenvironment interaction. JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.92056. eCollection 2017 May 18. PMID: 28515359; PMCID: PMC5436538.
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Nicotinamide metabolism regulates glioblastoma stem cell maintenance.

JCI insight

Jung J, Kim LJ, Wang X, Wu Q, Sanvoranart T, Hubert CG, Prager BC, Wallace LC, Jin X, Mack SC, Rich JN.
PMID: 28515364
JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.90019. eCollection 2017 May 18.

Metabolic dysregulation promotes cancer growth through not only energy production, but also epigenetic reprogramming. Here, we report that a critical node in methyl donor metabolism, nicotinamide N-methyltransferase (NNMT), ranked among the most consistently overexpressed metabolism genes in glioblastoma relative...

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Jung J, Kim LJ, Wang X, et al. Nicotinamide metabolism regulates glioblastoma stem cell maintenance. JCI Insight. 2017;2(10)doi: 10.1172/jci.insight.90019.
Jung, J., Kim, L. J., Wang, X., Wu, Q., Sanvoranart, T., Hubert, C. G., Prager, B. C., Wallace, L. C., Jin, X., Mack, S. C., & Rich, J. N. (2017). Nicotinamide metabolism regulates glioblastoma stem cell maintenance. JCI insight, 2(10), . https://doi.org/10.1172/jci.insight.90019
Jung, Jinkyu, et al. "Nicotinamide metabolism regulates glioblastoma stem cell maintenance." JCI insight vol. 2,10 (2017). doi: https://doi.org/10.1172/jci.insight.90019
Jung J, Kim LJ, Wang X, Wu Q, Sanvoranart T, Hubert CG, Prager BC, Wallace LC, Jin X, Mack SC, Rich JN. Nicotinamide metabolism regulates glioblastoma stem cell maintenance. JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.90019. eCollection 2017 May 18. PMID: 28515364; PMCID: PMC5436539.
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Ceramide synthesis regulates T cell activity and GVHD development.

JCI insight

Sofi MH, Heinrichs J, Dany M, Nguyen H, Dai M, Bastian D, Schutt S, Wu Y, Daenthanasanmak A, Gencer S, Zivkovic A, Szulc Z, Stark H, Liu C, Chang YJ, Ogretmen B, Yu XZ.
PMID: 28515365
JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.91701. eCollection 2017 May 18.

Allogeneic hematopoietic cell transplantation (allo-HCT) is an effective immunotherapy for a variety of hematologic malignances, yet its efficacy is impeded by the development of graft-versus-host disease (GVHD). GVHD is characterized by activation, expansion, cytokine production, and migration of alloreactive...

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Sofi MH, Heinrichs J, Dany M, et al. Ceramide synthesis regulates T cell activity and GVHD development. JCI Insight. 2017;2(10)doi: 10.1172/jci.insight.91701.
Sofi, M. H., Heinrichs, J., Dany, M., Nguyen, H., Dai, M., Bastian, D., Schutt, S., Wu, Y., Daenthanasanmak, A., Gencer, S., Zivkovic, A., Szulc, Z., Stark, H., Liu, C., Chang, Y. J., Ogretmen, B., & Yu, X. Z. (2017). Ceramide synthesis regulates T cell activity and GVHD development. JCI insight, 2(10), . https://doi.org/10.1172/jci.insight.91701
Sofi, M Hanief, et al. "Ceramide synthesis regulates T cell activity and GVHD development." JCI insight vol. 2,10 (2017). doi: https://doi.org/10.1172/jci.insight.91701
Sofi MH, Heinrichs J, Dany M, Nguyen H, Dai M, Bastian D, Schutt S, Wu Y, Daenthanasanmak A, Gencer S, Zivkovic A, Szulc Z, Stark H, Liu C, Chang YJ, Ogretmen B, Yu XZ. Ceramide synthesis regulates T cell activity and GVHD development. JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.91701. eCollection 2017 May 18. PMID: 28515365; PMCID: PMC5436544.
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Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps.

JCI insight

Kienhöfer D, Hahn J, Stoof J, Csepregi JZ, Reinwald C, Urbonaviciute V, Johnsson C, Maueröder C, Podolska MJ, Biermann MH, Leppkes M, Harrer T, Hultqvist M, Olofsson P, Munoz LE, Mocsai A, Herrmann M, Schett G, Holmdahl R, Hoffmann MH.
PMID: 28515366
JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.92920. eCollection 2017 May 18.

Many effector mechanisms of neutrophils have been implicated in the pathogenesis of systemic lupus erythematosus (SLE). Neutrophil extracellular traps (NETs) have been assigned a particularly detrimental role. Here we investigated the functional impact of neutrophils and NETs on a...

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Kienhöfer D, Hahn J, Stoof J, et al. Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps. JCI Insight. 2017;2(10)doi: 10.1172/jci.insight.92920.
Kienhöfer, D., Hahn, J., Stoof, J., Csepregi, J. Z., Reinwald, C., Urbonaviciute, V., Johnsson, C., Maueröder, C., Podolska, M. J., Biermann, M. H., Leppkes, M., Harrer, T., Hultqvist, M., Olofsson, P., Munoz, L. E., Mocsai, A., Herrmann, M., Schett, G., Holmdahl, R., & Hoffmann, M. H. (2017). Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps. JCI insight, 2(10), . https://doi.org/10.1172/jci.insight.92920
Kienhöfer, Deborah, et al. "Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps." JCI insight vol. 2,10 (2017). doi: https://doi.org/10.1172/jci.insight.92920
Kienhöfer D, Hahn J, Stoof J, Csepregi JZ, Reinwald C, Urbonaviciute V, Johnsson C, Maueröder C, Podolska MJ, Biermann MH, Leppkes M, Harrer T, Hultqvist M, Olofsson P, Munoz LE, Mocsai A, Herrmann M, Schett G, Holmdahl R, Hoffmann MH. Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps. JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.92920. eCollection 2017 May 18. PMID: 28515366; PMCID: PMC5436535.
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Glioblastoma-infiltrated innate immune cells resemble M0 macrophage phenotype.

JCI insight

Gabrusiewicz K, Rodriguez B, Wei J, Hashimoto Y, Healy LM, Maiti SN, Thomas G, Zhou S, Wang Q, Elakkad A, Liebelt BD, Yaghi NK, Ezhilarasan R, Huang N, Weinberg JS, Prabhu SS, Rao G, Sawaya R, Langford LA, Bruner JM, Fuller GN, Bar-Or A, Li W, Colen RR, Curran MA, Bhat KP, Antel JP, Cooper LJ, Sulman EP, Heimberger AB.
PMID: 26973881
JCI Insight. 2016;1(2). doi: 10.1172/jci.insight.85841. Epub 2016 Feb 25.

Glioblastomas are highly infiltrated by diverse immune cells, including microglia, macrophages, and myeloid-derived suppressor cells (MDSCs). Understanding the mechanisms by which glioblastoma-associated myeloid cells (GAMs) undergo metamorphosis into tumor-supportive cells, characterizing the heterogeneity of immune cell phenotypes within glioblastoma...

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Gabrusiewicz K, Rodriguez B, Wei J, et al. Glioblastoma-infiltrated innate immune cells resemble M0 macrophage phenotype. JCI Insight. 2016;1(2)doi: 10.1172/jci.insight.85841.
Gabrusiewicz, K., Rodriguez, B., Wei, J., Hashimoto, Y., Healy, L. M., Maiti, S. N., Thomas, G., Zhou, S., Wang, Q., Elakkad, A., Liebelt, B. D., Yaghi, N. K., Ezhilarasan, R., Huang, N., Weinberg, J. S., Prabhu, S. S., Rao, G., Sawaya, R., Langford, L. A., Bruner, J. M., Fuller, G. N., Bar-Or, A., Li, W., Colen, R. R., Curran, M. A., Bhat, K. P., Antel, J. P., Cooper, L. J., Sulman, E. P., & Heimberger, A. B. (2016). Glioblastoma-infiltrated innate immune cells resemble M0 macrophage phenotype. JCI insight, 1(2), . https://doi.org/10.1172/jci.insight.85841
Gabrusiewicz, Konrad, et al. "Glioblastoma-infiltrated innate immune cells resemble M0 macrophage phenotype." JCI insight vol. 1,2 (2016). doi: https://doi.org/10.1172/jci.insight.85841
Gabrusiewicz K, Rodriguez B, Wei J, Hashimoto Y, Healy LM, Maiti SN, Thomas G, Zhou S, Wang Q, Elakkad A, Liebelt BD, Yaghi NK, Ezhilarasan R, Huang N, Weinberg JS, Prabhu SS, Rao G, Sawaya R, Langford LA, Bruner JM, Fuller GN, Bar-Or A, Li W, Colen RR, Curran MA, Bhat KP, Antel JP, Cooper LJ, Sulman EP, Heimberger AB. Glioblastoma-infiltrated innate immune cells resemble M0 macrophage phenotype. JCI Insight. 2016;1(2). doi: 10.1172/jci.insight.85841. Epub 2016 Feb 25. PMID: 26973881; PMCID: PMC4784261.
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Characterization of candidate genes in inflammatory bowel disease-associated risk loci.

JCI insight

Peloquin JM, Goel G, Kong L, Huang H, Haritunians T, Sartor RB, Daly MJ, Newberry RD, McGovern DP, Yajnik V, Lira SA, Xavier RJ.
PMID: 27668286
JCI Insight. 2016 Aug 18;1(13):e87899. doi: 10.1172/jci.insight.87899.

GWAS have linked SNPs to risk of inflammatory bowel disease (IBD), but a systematic characterization of disease-associated genes has been lacking. Prior studies utilized microarrays that did not capture many genes encoded within risk loci or defined expression quantitative...

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Peloquin JM, Goel G, Kong L, et al. Characterization of candidate genes in inflammatory bowel disease-associated risk loci. JCI Insight. 2016;1(13):e87899doi: 10.1172/jci.insight.87899.
Peloquin, J. M., Goel, G., Kong, L., Huang, H., Haritunians, T., Sartor, R. B., Daly, M. J., Newberry, R. D., McGovern, D. P., Yajnik, V., Lira, S. A., & Xavier, R. J. (2016). Characterization of candidate genes in inflammatory bowel disease-associated risk loci. JCI insight, 1(13), e87899. https://doi.org/10.1172/jci.insight.87899
Peloquin, Joanna M, et al. "Characterization of candidate genes in inflammatory bowel disease-associated risk loci." JCI insight vol. 1,13 (2016): e87899. doi: https://doi.org/10.1172/jci.insight.87899
Peloquin JM, Goel G, Kong L, Huang H, Haritunians T, Sartor RB, Daly MJ, Newberry RD, McGovern DP, Yajnik V, Lira SA, Xavier RJ. Characterization of candidate genes in inflammatory bowel disease-associated risk loci. JCI Insight. 2016 Aug 18;1(13):e87899. doi: 10.1172/jci.insight.87899. PMID: 27668286; PMCID: PMC5033062.
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Enhanced protective antibody to a mutant meningococcal factor H-binding protein with low-factor H binding.

JCI insight

Granoff DM, Giuntini S, Gowans FA, Lujan E, Sharkey K, Beernink PT.
PMID: 27668287
JCI Insight. 2016 Sep 08;1(14):e88907. doi: 10.1172/jci.insight.88907.

Meningococcal factor H-binding protein (FHbp) is an antigen in 2 serogroup B meningococcal vaccines. FHbp specifically binds human and some nonhuman primate complement FH. To investigate the effect of binding of FH to FHbp on protective antibody responses, we...

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Granoff DM, Giuntini S, Gowans FA, et al. Enhanced protective antibody to a mutant meningococcal factor H-binding protein with low-factor H binding. JCI Insight. 2016;1(14):e88907doi: 10.1172/jci.insight.88907.
Granoff, D. M., Giuntini, S., Gowans, F. A., Lujan, E., Sharkey, K., & Beernink, P. T. (2016). Enhanced protective antibody to a mutant meningococcal factor H-binding protein with low-factor H binding. JCI insight, 1(14), e88907. https://doi.org/10.1172/jci.insight.88907
Granoff, Dan M, et al. "Enhanced protective antibody to a mutant meningococcal factor H-binding protein with low-factor H binding." JCI insight vol. 1,14 (2016): e88907. doi: https://doi.org/10.1172/jci.insight.88907
Granoff DM, Giuntini S, Gowans FA, Lujan E, Sharkey K, Beernink PT. Enhanced protective antibody to a mutant meningococcal factor H-binding protein with low-factor H binding. JCI Insight. 2016 Sep 08;1(14):e88907. doi: 10.1172/jci.insight.88907. PMID: 27668287; PMCID: PMC5033880.
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MPEG1/perforin-2 mutations in human pulmonary nontuberculous mycobacterial infections.

JCI insight

McCormack RM, Szymanski EP, Hsu AP, Perez E, Olivier KN, Fisher E, Goodhew EB, Podack ER, Holland SM.
PMID: 28422754
JCI Insight. 2017 Apr 20;2(8). doi: 10.1172/jci.insight.89635. eCollection 2017 Apr 20.

Perforin-2 is a highly conserved pore-forming protein encoded by macrophage expressed gene 1 (MPEG1). A number of studies have shown that Perforin-2-deficient mice are unable to survive following a bacterial challenge that is nonlethal in WT mice. There is...

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McCormack RM, Szymanski EP, Hsu AP, et al. MPEG1/perforin-2 mutations in human pulmonary nontuberculous mycobacterial infections. JCI Insight. 2017;2(8)doi: 10.1172/jci.insight.89635.
McCormack, R. M., Szymanski, E. P., Hsu, A. P., Perez, E., Olivier, K. N., Fisher, E., Goodhew, E. B., Podack, E. R., & Holland, S. M. (2017). MPEG1/perforin-2 mutations in human pulmonary nontuberculous mycobacterial infections. JCI insight, 2(8), . https://doi.org/10.1172/jci.insight.89635
McCormack, Ryan M, et al. "MPEG1/perforin-2 mutations in human pulmonary nontuberculous mycobacterial infections." JCI insight vol. 2,8 (2017). doi: https://doi.org/10.1172/jci.insight.89635
McCormack RM, Szymanski EP, Hsu AP, Perez E, Olivier KN, Fisher E, Goodhew EB, Podack ER, Holland SM. MPEG1/perforin-2 mutations in human pulmonary nontuberculous mycobacterial infections. JCI Insight. 2017 Apr 20;2(8). doi: 10.1172/jci.insight.89635. eCollection 2017 Apr 20. PMID: 28422754; PMCID: PMC5396519.
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β-catenin and PI3Kδ inhibition expands precursor Th17 cells with heightened stemness and antitumor activity.

JCI insight

Majchrzak K, Nelson MH, Bowers JS, Bailey SR, Wyatt MM, Wrangle JM, Rubinstein MP, Varela JC, Li Z, Himes RA, Chan SS, Paulos CM.
PMID: 28422756
JCI Insight. 2017 Apr 20;2(8). doi: 10.1172/jci.insight.90547. eCollection 2017 Apr 20.

ICOS costimulation generates Th17 cells with durable memory responses to tumor. Herein, we found that ICOS induces PI3K/p110δ/Akt and Wnt/β-catenin pathways in Th17 cells. Coinhibiting PI3Kδ and β-catenin altered the biological fate of Th17 cells. Th17 cells inhibited of...

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Majchrzak K, Nelson MH, Bowers JS, et al. β-catenin and PI3Kδ inhibition expands precursor Th17 cells with heightened stemness and antitumor activity. JCI Insight. 2017;2(8)doi: 10.1172/jci.insight.90547.
Majchrzak, K., Nelson, M. H., Bowers, J. S., Bailey, S. R., Wyatt, M. M., Wrangle, J. M., Rubinstein, M. P., Varela, J. C., Li, Z., Himes, R. A., Chan, S. S., & Paulos, C. M. (2017). β-catenin and PI3Kδ inhibition expands precursor Th17 cells with heightened stemness and antitumor activity. JCI insight, 2(8), . https://doi.org/10.1172/jci.insight.90547
Majchrzak, Kinga, et al. "β-catenin and PI3Kδ inhibition expands precursor Th17 cells with heightened stemness and antitumor activity." JCI insight vol. 2,8 (2017). doi: https://doi.org/10.1172/jci.insight.90547
Majchrzak K, Nelson MH, Bowers JS, Bailey SR, Wyatt MM, Wrangle JM, Rubinstein MP, Varela JC, Li Z, Himes RA, Chan SS, Paulos CM. β-catenin and PI3Kδ inhibition expands precursor Th17 cells with heightened stemness and antitumor activity. JCI Insight. 2017 Apr 20;2(8). doi: 10.1172/jci.insight.90547. eCollection 2017 Apr 20. PMID: 28422756; PMCID: PMC5396523.
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Therapeutic inhibition of soluble brain TNF promotes remyelination by increasing myelin phagocytosis by microglia.

JCI insight

Karamita M, Barnum C, Möbius W, Tansey MG, Szymkowski DE, Lassmann H, Probert L.
PMID: 28422748
JCI Insight. 2017 Apr 20;2(8). doi: 10.1172/jci.insight.87455. eCollection 2017 Apr 20.

Multiple sclerosis (MS) is an inflammatory CNS demyelinating disease in which remyelination largely fails. Transmembrane TNF (tmTNF) and TNF receptor 2 are important for remyelination in experimental MS models, but it is unknown whether soluble TNF (solTNF), a major...

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Karamita M, Barnum C, Möbius W, et al. Therapeutic inhibition of soluble brain TNF promotes remyelination by increasing myelin phagocytosis by microglia. JCI Insight. 2017;2(8)doi: 10.1172/jci.insight.87455.
Karamita, M., Barnum, C., Möbius, W., Tansey, M. G., Szymkowski, D. E., Lassmann, H., & Probert, L. (2017). Therapeutic inhibition of soluble brain TNF promotes remyelination by increasing myelin phagocytosis by microglia. JCI insight, 2(8), . https://doi.org/10.1172/jci.insight.87455
Karamita, Maria, et al. "Therapeutic inhibition of soluble brain TNF promotes remyelination by increasing myelin phagocytosis by microglia." JCI insight vol. 2,8 (2017). doi: https://doi.org/10.1172/jci.insight.87455
Karamita M, Barnum C, Möbius W, Tansey MG, Szymkowski DE, Lassmann H, Probert L. Therapeutic inhibition of soluble brain TNF promotes remyelination by increasing myelin phagocytosis by microglia. JCI Insight. 2017 Apr 20;2(8). doi: 10.1172/jci.insight.87455. eCollection 2017 Apr 20. PMID: 28422748; PMCID: PMC5396518.
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